Please use this identifier to cite or link to this item: http://earsiv.odu.edu.tr:8080/xmlui/handle/11489/3244
Full metadata record
DC FieldValueLanguage
dc.contributor.authorSunman, H.-
dc.contributor.authorOzkan, A.-
dc.contributor.authorCanpolat, U.-
dc.contributor.authorBayrak, T.-
dc.contributor.authorYorgun, H.-
dc.contributor.authorSahiner, L.-
dc.contributor.authorKaya, B.-
dc.contributor.authorOzkara, A.-
dc.contributor.authorAytemir, K.-
dc.contributor.authorOto, A.-
dc.date.accessioned2022-09-09T10:31:46Z-
dc.date.available2022-09-09T10:31:46Z-
dc.date.issued2013-
dc.identifier.urihttp://doi.org/10.1093/eurheartj/eht309.P3184-
dc.identifier.urihttp://earsiv.odu.edu.tr:8080/xmlui/handle/11489/3244-
dc.description.abstractThe aim of the present study was to evaluate the influence of cardiac resynchronization therapy (CRT) on systemic inflammation, oxidative stress, extracellular-matrix remodeling, neurohormonal pathways, myocyte injury and stress for the pathophysiology of chronic heart failure.en_US
dc.language.isoengen_US
dc.publisherOXFORD UNIV PRESSGREAT CLARENDON ST, OXFORD OX2 6DP, ENGLANDen_US
dc.relation.isversionof10.1093/eurheartj/eht309.P3184en_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.subjectoxidative stress chronic heart failure endothelin-1 echocardiographyen_US
dc.titleThe influence of cardiac resynchronization therapy on molecular pathways in pathophysiology of heart failureen_US
dc.typearticleen_US
dc.relation.journalEUROPEAN HEART JOURNALen_US
dc.contributor.departmentOrdu Üniversitesien_US
dc.contributor.authorID0000-0002-4250-1706en_US
dc.identifier.volume34en_US
dc.identifier.startpage586en_US
dc.identifier.endpage586en_US
Appears in Collections:Temel Tıp Bilimleri

Files in This Item:
There are no files associated with this item.


Items in DSpace are protected by copyright, with all rights reserved, unless otherwise indicated.